Insight
Women’s HealthX launches in Boston this December to transform women’s health through data and science

On December 3–4, 2026 in Boston, Women’s HealthX (WoHX) will bring together 750 global leaders from pharma and biotech, hospitals and health systems, health insurers, employers, investors, startups, and government, all actively seeking proven technologies, data, and partners to advance women’s health care, research, and outcomes across the life course.
WoHX is the number one event in women’s health, unifying the full lifecycle of female healthcare through data, science, and evidence-based innovation to close the sex difference data gap and drive better clinical outcomes for women worldwide
Unlike any other event, WoHX goes beyond discussion to focus on implementation of representative data sets to drive meaningful change. The exhibition directly addresses the conditions that affect women differently and disproportionately, across every stage of life.
Attendees will gain clear insight into which areas are overhyped versus underfunded, where the biggest evidence gaps remain, and how data, science, and evidence can drive measurable change in policy, reimbursement, product development, and clinical practice.
Julie Rios, Division Director, Reproductive Endocrinology & Infertility at UPMC, shared why she is looking forward to attending:
She said: “I’m looking forward to connecting with innovators across women’s health to explore new technologies, collaborations, and care models that can help us solve our most complex reproductive health cases and improve outcomes for patients who currently have limited options.”
Taking place in Boston, the global hub for healthcare innovation, research, and medical institutions, whose collaborative ecosystem aligns perfectly with WoHX’s mission to accelerate the adoption of clinical solutions, and improve outcomes for women worldwide.
Across seven dedicated stages spanning Evidence, Data & Innovation, Fertility & Reproductive Health, Menopause & Healthy Aging, Maternity & Maternal Care, Sexual Health & Wellness, Cognitive Health & Wellness, and Chronic Disease Management, attendees will benefit from:
- 100+ hours of free education from 150+ expert speakers
- Direct access to senior decision-makers and key industry leaders
- Tailored one-to-one meetings with solution providers across medical devices, CROs, and analytics software
- Hands-on exploration of AI-powered tools, digital therapeutics, wearables, telehealth, and integrated care models via the interactive HealthXpo floor, featuring live demonstrations and hands-on clinical showcases
- The Women’s Health Startup Zone, connecting founders directly with investors
- The Career Zone, linking attendees with postgraduate programs, universities, and research centres, alongside masterclasses in AI literacy, data analytics, and research innovation
Early confirmed speakers include:
- Michael Annichine, CEO, Magee-Womens Research Institute and Foundation
- David Friend, Chief Science Officer, Daré Bioscience
- Emily Lau, Director, Women’s Heart Health Program, Brigham and Women’s Hospital
- Carolee Lee, CEO & Founder, WHAM
- Suneela Vegunta, Vice Chair, Women’s Health Research Division, Mayo Clinic
- Barb DePree, Director of Women’s Health, Holland Hospital
- Jodi Neuhauser, Founder & CEO, In Women’s Health
- Julie Rios, Division Director, Reproductive Endocrinology & Infertility, UPMC
- Kesha O’Reilly, Global Director, Medical Affairs HIV Franchise, Gilead Sciences
- Katie Baca-Motes, CEO GSD, Health Research
- Catherine Monk, Founding Director, Center for the Transition to Parenthood
- Mitzi Krockover, CEO & Founder, WomanCentered
Further announcements, including speaker confirmations and agenda highlights, will be released in the coming months.
Because Women’s HealthX believes in healthcare equity, attendance is free for practitioners within pharma, biotech, corporate enterprises, and medical officers and leaders within hospital and healthcare systems.
Register your free place now: https://www.alphaevents.com/events-whx/srspricing#/?utm_source=FemTechW&utm_medium=Media%20Partner&utm_campaign=52531.001%20-%20WHX%20-%20MP%20-%20FemTech%20World%20-%20Press%20Release&utm_term=&utm_content=&disc=&extTreatId=7631829
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Common cancer marker may play active role in preventing the disease, study finds

Ki-67, a protein used to measure tumour growth, may also help prevent chromosome errors that drive cancer, a study suggests.
The findings could change how scientists view Ki-67, a marker commonly used in breast cancer and other tumours to assess how quickly cancer cells are growing.
Researchers found the protein may help preserve genome stability by maintaining the structural integrity of centromeres, key parts of chromosomes that help ensure DNA is shared correctly during cell division.
The research was led by professor Paola Vagnarelli at Brunel University of London in collaboration with scientists at the University of Edinburgh and the Technical University of Berlin.
Professor Vagnarelli said: “Doctors already measure Ki-67 to see how aggressive a cancer might be. But our results suggest it is actually helping maintain genome stability.
“That means it may be more than a marker. It could potentially also be a therapeutic target.”
The study examined three proteins that attach to chromosomes during cell division and help rebuild the molecular system that tells each new cell what kind of cell it is.
Every human cell carries identical DNA. What makes a liver cell different from a brain cell is which genes are switched on and which are kept inactive.
When a cell divides, that entire system of switches must be rebuilt. The three proteins involved in this process were Ki-67, Repo-Man and PNUTS.
Vagnarelli’s team developed a method that individually removes each protein from a living cell at the precise point of division. Older techniques could not isolate that moment cleanly.
They found that cells rely on all three proteins to reset themselves after division, but each failed in a different way when removed.
Without PNUTS, gene activity spiralled out of control and thousands of genes switched on at once.
Without Repo-Man, cells escaped safety checkpoints that usually stop damaged or abnormal cells from continuing to divide.
“What we didn’t expect was how clean the separation was,” said Vagnarelli.
Each protein fails in its own specific way. There is no redundancy, no safety net. Which means there are three separate points at which this process can go wrong.
“When the system breaks down, cells can emerge with the wrong number of chromosomes. That condition, called aneuploidy, is seen in disorders such as Down syndrome and in many cancers.
“We also found that these chromosome errors can trigger inflammatory signals inside the cell.”
Aneuploidy means a cell has too many or too few chromosomes, which can disrupt normal growth and function.
Inflammatory signals are chemical messages that can make a cell behave as if it is responding to injury or infection.
“These cells behave almost as if they are under attack,” said Vagnarelli.
“The immune response switches on because the genome is unstable.
“That link between chromosome imbalance and inflammation could help explain patterns we see in several diseases.”
The researchers said the findings may help cancer scientists better understand how chromosome instability, loss of gene regulation and cells dividing before they are ready contribute to tumour growth.
They said understanding the normal machinery that prevents these errors may help researchers find ways to push cancer cells into making mistakes they cannot survive.
“We now have a clearer map of the machinery that resets the cell after division,” said Vagnarelli.
“That knowledge gives us a starting point for thinking about new therapeutic approaches.”
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