News
Breastfeeding support app LatchAid rebrands to Anya
The app has benefitted from a complete redesign and redevelopment to provide clear visual learning for latching
The breastfeeding support app LatchAid has rebranded to become Anya, aiming to reach more parents across the globe.
The developers say the rebrand will allow the app to expand its scope beyond breastfeeding to the first 1,001 days of parenthood, helping families to overcome a much broader range of challenges in line with the UK Government’s A Better Start in Life initiative.
The LatchAid name will live on as the 3D interactive animation tool coaches in the app.
Anya aims to empower new parents to get access to support and information and allow healthcare professionals to provide continuity of care, bridging the gap between antenatal and postnatal care.
The company says the app has benefitted from a complete redesign and redevelopment to provide clear visual learning for latching.
Other components will offer articles, videos, webinars, and content to parents across the whole pregnancy, parenting and infant feeding spectrum while virtual community groups have been expanded.
After proving that the app can double the average exclusive breastfeeding rate postnatally at six-eight weeks in a recent NHS Pilot, the team have worked on bringing the new app to market, partnering with a number of Integrated Care Systems and NHS Trusts.

According to the company’s data, the app is already proving its worth in selected Integrated Care Systems, supporting continuity of care, improved patient outcomes, cost of care reductions and bridges the breastfeeding support inequalities gap.
LatchAid has recently won the coveted SBRI Healthcare funding competition to develop additional features and content in the app to tackle healthcare inequalities in maternity care.
This project will be co-developed with key NHS partners, along with groups of parents in target demographics to address health inequalities and support underserved communities while improving wider breastfeeding rates.
“It’s been so exciting to see my baby LatchAid grow up into Anya, on both iOS and Android,” said Dr Chen Mao Davies, LatchAid founder and CEO.
“We know how important access to evidence-based information and support has already been to new parents via the app. We look forward to reaching many more families inclusively with our SBRI Healthcare funding.”
Diagnosis
Lung cancer drug shows breast cancer potential
Ovarian cancer cells quickly activate survival responses after PARP inhibitor treatment, and a lung cancer drug could help block this, research suggests.
PARP inhibitors are a common treatment for ovarian cancer, particularly in tumours with faulty DNA repair. They stop cancer cells fixing DNA damage, which leads to cell death, but many tumours later stop responding.
Researchers identified a way cancer cells may survive PARP inhibitor treatment from the outset, pointing to a potential way to block that response. A Mayo Clinic team found ovarian cancer cells rapidly switch on a pro-survival programme after exposure to PARP inhibitors. A key driver is FRA1, a transcription factor (a protein that turns genes on and off) that helps cancer cells adapt and avoid death.
The team then tested whether brigatinib, a drug approved for certain lung cancers, could block this response and boost the effect of PARP inhibitors. Brigatinib was chosen because it inhibits multiple signalling pathways involved in cancer cell survival.
In laboratory studies, combining brigatinib with a PARP inhibitor was more effective than either treatment alone. Notably, the effect was seen in cancer cells but not normal cells, suggesting a more targeted approach.
Brigatinib also appeared to act in an unexpected way. Rather than working through the usual DNA repair routes, it shut down two signalling molecules, FAK and EPHA2, that aggressive ovarian cancer cells rely on. FAK and EPHA2 are proteins that relay survival signals inside cells. Blocking both at once weakened the cells’ ability to adapt and resist treatment, making them more vulnerable to PARP inhibitors.
Tumours with higher levels of FAK and EPHA2 responded better to the drug combination. Other data link high levels of these molecules to more aggressive disease, pointing to potential benefit in harder-to-treat cases.
Arun Kanakkanthara, an oncology investigator at Mayo Clinic and a senior author of the study, said: “This work shows that drug resistance does not always emerge slowly over time; cancer cells can activate survival programmes very early after treatment begins.”
John Weroha, a medical oncologist at Mayo Clinic and a senior author of the study, said: “From a clinical perspective, resistance remains one of the biggest challenges in treating ovarian cancer. By combining mechanistic insights from Dr Kanakkanthara’s laboratory with my clinical experience, this preclinical work supports the strategy of targeting resistance early, before it has a chance to take hold. This strategy could improve patient outcomes.”
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