Scale-IT-up Workshop 2026 – Advancing Digital Women’s Health
Scale-IT-up Workshop 2026 – Advancing Digital Women’s Health
The 7th Scale-IT-Up Workshop 2026 will be held on 2–3 March 2026 in Marbella, Spain, in conjunction with the 19th International Joint Conference on Biomedical Engineering Systems and Technologies – BIOSTEC 2026.
This year’s theme, Advancing Digital Women’s Health Across the Life Course, explores how digital technologies—including apps, sensors, wearables, and AI—can address female-specific conditions (such as endometriosis, PCOS, menopause, and maternal health) as well as conditions disproportionately affecting women (e.g., cardiovascular disease, migraine, mental health).
Key Topics:
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Closing the gender data gap in women’s health through scalable digital tools
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Inclusive, intersectional, and equitable digital health interventions
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Real-world implementation of wearables, self-tracking, and monitoring technologies
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Business models, reimbursement pathways, and health system integration
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Methodological approaches for equitable, sex- and gender-disaggregated data collection
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Scaling innovations across populations and healthcare systems
Who Should Attend:
Academics, clinicians, innovators, policymakers, data scientists, public health experts, and industry partners working at the intersection of women’s health, digital health, and equity.
Workshop Chairs:
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Dr. Marcia Nißen, University of Zurich, University of St. Gallen & ETH Zurich
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Prof. Dr. Hannes Schlieter, TU Dresden
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Prof. Dr. Tobias Kowatsch, University of Zurich, University of St. Gallen & ETH Zurich
Important Deadlines:
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Paper Submission: November 20, 2025
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Authors Notification: January 8, 2026
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Camera Ready: January 22, 2026
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Registration: January 22, 2026
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Workshop: March 2–3, 2026
Organizer: Centre for Digital Health Interventions (University of Zurich, University of St. Gallen, ETH Zurich)
FT World does not take responsibility for any changes to this event, which we have published in good faith. Please direct any queries to the organiser.
Diagnosis
Lung cancer drug shows breast cancer potential
Ovarian cancer cells quickly activate survival responses after PARP inhibitor treatment, and a lung cancer drug could help block this, research suggests.
PARP inhibitors are a common treatment for ovarian cancer, particularly in tumours with faulty DNA repair. They stop cancer cells fixing DNA damage, which leads to cell death, but many tumours later stop responding.
Researchers identified a way cancer cells may survive PARP inhibitor treatment from the outset, pointing to a potential way to block that response. A Mayo Clinic team found ovarian cancer cells rapidly switch on a pro-survival programme after exposure to PARP inhibitors. A key driver is FRA1, a transcription factor (a protein that turns genes on and off) that helps cancer cells adapt and avoid death.
The team then tested whether brigatinib, a drug approved for certain lung cancers, could block this response and boost the effect of PARP inhibitors. Brigatinib was chosen because it inhibits multiple signalling pathways involved in cancer cell survival.
In laboratory studies, combining brigatinib with a PARP inhibitor was more effective than either treatment alone. Notably, the effect was seen in cancer cells but not normal cells, suggesting a more targeted approach.
Brigatinib also appeared to act in an unexpected way. Rather than working through the usual DNA repair routes, it shut down two signalling molecules, FAK and EPHA2, that aggressive ovarian cancer cells rely on. FAK and EPHA2 are proteins that relay survival signals inside cells. Blocking both at once weakened the cells’ ability to adapt and resist treatment, making them more vulnerable to PARP inhibitors.
Tumours with higher levels of FAK and EPHA2 responded better to the drug combination. Other data link high levels of these molecules to more aggressive disease, pointing to potential benefit in harder-to-treat cases.
Arun Kanakkanthara, an oncology investigator at Mayo Clinic and a senior author of the study, said: “This work shows that drug resistance does not always emerge slowly over time; cancer cells can activate survival programmes very early after treatment begins.”
John Weroha, a medical oncologist at Mayo Clinic and a senior author of the study, said: “From a clinical perspective, resistance remains one of the biggest challenges in treating ovarian cancer. By combining mechanistic insights from Dr Kanakkanthara’s laboratory with my clinical experience, this preclinical work supports the strategy of targeting resistance early, before it has a chance to take hold. This strategy could improve patient outcomes.”
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